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OxyContin is a semi-synthetic opiate prescribed to reduce medium to severe pain. The UK is currently facing an opioid crisis due to overprescribing powerful opiate and opioid analgesic drugs such as OxyContin. The current painkiller epidemic is particularly evident in the north of England, where research has shown that painkilling opiates are prescribed up to four times more than in London.
OxyContin itself is a drug that holds huge potential for abuse and is extremely addictive. If you or a loved one are suffering from OxyContin addiction, we urge you to seek professional addiction help without delay.
OxyContin is a potent semi-synthetic opioid pain killer that belongs to a class of prescription medications known as opioid narcotic analgesics. It is usually only prescribed for severe pain and is most commonly used to control the pain in cancer or post-operative pain.
The makers of OxyContin laid off their entire sales team following more than 2,000 allegations and lawsuits from cities, states and counties in the US. Sales of OxyContin has been alleged to have fueled the US opioid crisis.
OxyContin is derived from the opium poppy plant. It works on the brain’s opioid receptors to change how the body interprets and responds to pain, providing effective pain relief.
The higher strengths of OxyContin should only be used if a person is already used to the medication. Strengths of 40 milligrams or more can cause an overdose and death in someone who is not tolerant of opiates.
OxyContin immediate-release tablets start to work within 15 to 30 minutes from ingestion. The peak pharmaceutical effects can be felt within 1 hour, and OxyContin’s effects last between 3 to 6 hours, depending on the individual’s pain levels and opiate tolerance.
OxyContin is only available by prescription from a physician. It comes in various doses in tablet form, and is often prescribed to those who suffer chronic pain. OxyContin was originally approved by the Food and Drug Administration in 1995, meaning it’s still one of the newer painkillers on the market
OxyContin comes in various forms and is often combined with other analgesics for more effective pain relief. OxyContin is available in the following forms in the UK:
OxyContin immediate-release tablets and capsules (brand names include OxyContin, Oxaydo, OxyFast, OxyIR, OxyNorm, Roxicodone and Roxybond)
OxyContin oral solution – Used for individuals that are unable to swallow tablets and also for breakthrough pain management in cancer patients. OxyContin solution can also be administered intravenously through a tube.
OxyContin sublingual (under the tongue administration)
OxyContin extended-release tablets and capsules – OxyContin ER and Xtampza ER –extended-release gradually releases a steady supply of OxyContin into the bloodstream over a period of 12 hours
Immediate-release OxyContin with paracetamol (brand names include Percocet, Endocet, Roxicet sand Tylox)
Immediate-release OxyContin with aspirin (brand names include Endodan, Oxycodan, Percodan and Roxiprin) Immediate-release OxyContin with ibuprofen (Combunox)
Extended-release OxyContin with Naloxone (brand names include Targin, Targiniq and
Targinact) works for 10-12 hours of controlled release.
Extended-release OxyContin with naltrexone (Troxyca) works for 10-12 hours of controlled release.
Lightheadedness
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OxyContin (oxycodone hydrochloride) is an opioid drug used for the management of moderate to severe pain, usually for an extended time period. Oxycontin is not an "as needed for pain (PRN) drug." Oxycontin is available as a generic drug.
Management of pain severe enough to require daily, around-the-clock, long-term analgesia for which alternative opioid therapies are inadequate in adults and opioid-tolerant (already on ≥20mg/day oral oxycodone or its equivalent) children.
Recently, there has been a lot of media focus on this prescription drug due to increasing reports of its abuse. According to an Office ofNational Drug Control Policy (ONDCP) fact sheet, an estimated 1.6 million Americans used prescription-type pain relievers for non-medical reasons for the first time in 1998. Furthermore, ONDCP reports that the number of oxycodone emergency cases increased nearly 36 percent in a single year, from 3,369 in January to June 1999 to 5,261 in January to June 2000.
Assess the potential need for access to naloxone when initiating and renewing therapy. Consider prescribing naloxone based on risk factors for overdose (eg, history of opioid use disorder, prior opioid overdose, household members or other close contacts at risk for accidental ingestion or overdose). Abuse potential (monitor). Increased risk of fatal respiratory depression (esp. when initiating therapy and during dose increases); monitor.
Accidental exposure may result in fatal overdose (esp. children). Sleep-related breathing disorders (including central sleep apnea (CSA), sleep-related hypoxemia); consider dose reduction if CSA develops. Risk of neonatal opioid withdrawal syndrome. Pulmonary disease (eg, COPD, cor pulmonale); monitor for respiratory depression (esp. within the first 24–72hrs of initiating therapy and after dose increases); consider alternative non-opioid analgesics. Head injury. Impaired consciousness, coma, shock; avoid. Increased intracranial pressure, brain tumors; monitor. Convulsive disorders. Difficulty swallowing. Underlying GI disorders (eg, esophageal or colon cancer with a small GI lumen). Biliary tract disease.
Acute pancreatitis.
Acute alcoholism. Drug abusers. Avoid abrupt cessation. Reevaluate periodically. Impaired renal or hepatic function. Elderly. Cachectic. Debilitated. Pregnancy. Labor & delivery, nursing mothers: not recommended.
Increased risk of hypotension, respiratory depression, sedation with benzodiazepines or other CNS depressants (eg, non-benzodiazepine sedatives/hypnotics, anxiolytics, general anesthetics, phenothiazines, tranquilizers, muscle relaxants, antipsychotics, alcohol, other opioids); reserve concomitant use in those for whom alternative options are inadequate; limit dosages/durations to minimum required; monitor closely; consider prescribing naloxone if concomitant use is warranted.
During or within 14 days of MAOIs: not recommended. Risk of serotonin syndrome with serotonergic drugs (eg, SSRIs, SNRIs, TCAs, triptans, 5HT3 receptor antagonists, mirtazapine, trazodone, tramadol, cyclobenzaprine, metaxalone, MAOIs, linezolid, IV methylene blue); monitor and discontinue if suspected. Avoid mixed agonist/antagonist opioids (eg, butorphanol, nalbuphine, pentazocine) or partial agonist (eg, buprenorphine); may reduce effects and/or precipitate withdrawal symptoms.
May be potentiated by CYP3A4 inhibitors (eg, macrolides, azole antifungals, protease inhibitors). May be antagonized by CYP3A4 inducers (eg, rifampin, carbamazepine, phenytoin). May antagonize diuretics. Paralytic ileus may occur with anticholinergics. May increase serum amylase
Swallow whole. Individualize. Usually given on a 12hr schedule. ≥18yrs: Opioid-naive, opioid non-tolerant: initially 10mg every 12hrs. May increase total daily dose by 25–50%; adjust at 1–2 day intervals. Conversion from other opioids or combinations: see full labeling. Elderly (debilitated and opioid non-tolerant), hepatic dysfunction or concomitant CNS depressants: initiate at ⅓ to ½ the usual starting dose and titrate slowly. 60mg and 80mg tabs, a single dose >40mg, or a total daily dose >80mg: for use in opioid-tolerant patients only. Withdraw gradually (esp. if opioid-dependent), taper by ≤10–25% every 2–4 weeks. See full labeling.
Not for use as an as-needed (prn) analgesic. Use only if alternative treatment options (eg, non-opioid analgesics, immediate-release opioids) are ineffective, not tolerated, or otherwise inadequate to provide sufficient management of pain.
Significant respiratory depression. Acute or severe bronchial asthma in an unmonitored setting or in the absence of resuscitative equipment. Known or suspected paralytic ileus or GI obstruction.
Addiction, abuse, and misuse. Risk evaluation and mitigation strategy (REMS). Life-threatening respiratory depression. Accidental ingestion. Neonatal opioid withdrawal syndrome. Cytochrome P450 3A4 interaction. Risks from concomitant use with benzodiazepines or other CNS depressants.
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